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Incidence
of Immune Reconstitution Syndrome in HIV-Tuberculosis Coinfected
Patients in India
HAART
has led to dramatic reductions in HIV-related morbidity
and mortality. HAART suppresses viral replication, allowing
for partial restoration of the immune system and protection against
opportunistic
pathogens.
Immune
reconstitution, however, can result in an inflammatory response
against infectious and non-infectious antigens and an apparent clinical
deterioration of the patient. This phenomenon has been described
under various descriptions such as "paradoxical reaction,"
"immune restoration
disease," "HAART
attacks," "immune
reconstitution syndrome (IRS)," and "immune
reconstitution inflammatory syndrome."
Previous
reports of IRS have primarily been published in the developed world.
With declining costs in antiretroviral drugs, and production by
generic manufacturers, care centers in the developing world are
now able to afford and administer antiretroviral therapy to HIV
patients.
With
increased coprevalence of opportunistic infections and access to
antiretroviral therapy in the developing world, researchers conducted
this study to determine the incidence of immune reconstitution in
resource-limited settings and the implications for clinicians.
In
this study, 11 of 144 HIV and tuberculosis
(TB)-coinfected individuals followed for 72 person-years
developed IRS within 6 months of initiating generic HAART.
All
of the IRS patients were male, with a median age of 29 years; median
CD4 at HAART initiation was 123 cells/mm3, and 6-month median CD4
rise was 124 cells/mm3.
There
was no statistical difference in CD4 rise or CD4 count and duration
of TB treatment at HAART initiation between those who did and those
who did not develop IRS (P = 0.8380).
The
median time to development of clinical IRS was 42 days (range 10-89
days).
The
incidence of IRS in this cohort is 15.2 cases per 100 patient-years.
With
increased coprevalence of opportunistic infections, especially TB,
and increasing access to antiretroviral therapy in the developing
world, clinicians in these countries must be able to identify IRS
and relieve symptoms without compromising clinical care.
Discussion
Unfortunately,
the human host's enhanced ability to mount an immune reaction and
protect from the destruction of infectious agents may itself cause
morbidity to the host. There have been cases published of clinical
"progression" despite adequate anti-TB and antiretroviral
therapy, including prolonged fever, increasing respiratory symptoms,
increasing lymphadenopathy, ascites, and growth of intracranial
tuberculomas.
In
this cohort from a resource-limited setting with a high background
rate of TB, there is a high incidence of IRS. As the use of antiretroviral
therapy increases in the developing world, initiation of therapy
while patients have active opportunistic infections will lead to
significant morbidity. An association between the duration of initiation
of TB treatment and HAART initiation was seen in an earlier study
for the occurrence of IRS. This association was not seen in our
series. Appropriate therapy must be maintained in the face of apparently
worsening clinical symptoms. The use of corticosteroids has anecdotally
been successful in decreasing IRS without significantly compromising
clinical care.
However,
there are no definitive guidelines for care of the patient with
IRS. At this center in India, patients are counseled on the condition
and are advised to continue with treatment. All patients are currently
stable and tolerating therapy with no further adverse events, according
to the authors.
In
conclusion, the authors write, “With reports of IRS growing in frequency,
and now detected significantly in a tertiary HIV center in the developing
world, clinical trials in developing countries need to be conducted
to help physicians better understand when to initiate antiretroviral
therapy in the context of opportunistic infections.
“In
addition, without adequate guidelines to identify and treat IRS,
clinicians treating HIV-infected individuals in the developing world
will face a new set of challenges to safe and effective therapy.”
02/11/05
Reference
N
Kumarasamy and others. Incidence of Immune Reconstitution Syndrome in HIV/Tuberculosis-Coinfected
Patients After Initiation of Generic Antiretroviral Therapy in India.
Journal of Acquired Immune Deficiency Syndromes 37(5): 1574-1576,
December 15, 2004.
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