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Use
of Prednisolone in HIV-associated Tuberculous Pleurisy Is Not Recommended
Active
tuberculosis
may accelerate progression
of HIV-1 infection by promoting viral
replication in activated lymphocytes. Glucocorticoids are used in pleural * tuberculosis to reduce
inflammation-induced pathology, and their use also
might reduce progression of HIV by suppressing immune
activation. Researchers examined the effect that prednisolone has on survival in HIV-1 associated
pleural tuberculosis.
The
investigators conducted a randomized, double-blind, placebo-controlled trial of prednisolone as an adjunct to tuberculosis treatment, in adults with HIV-1 associated pleural
tuberculosis. The primary outcome was death. Analysis
was by intention to treat.
Results
Of
197 participants, 99 were assigned to the
prednisolone group and 98 to the placebo group.
The mortality rate was 21 deaths/100 person-years (pyr) in the prednisolone group and 25 deaths/100 pyr in the placebo group (age-, sex-, and initial CD4+ T cell count adjusted mortality
rate ratio, 0.99 [95% confidence interval, 0.62 1.56] [P
= .95]).
Resolution
of tuberculosis was faster in the prednisolone group,
but recurrence rates were slightly (though not significantly)
higher, and use of prednisolone was associated with
a significantly higher incidence of Kaposi
sarcoma (4.2 cases/100 pyr, compared with 0 cases/100 pyr [P = .02]).
Conclusions
The
authors conclude, “In view of the lack of survival
benefit and the increased risk of Kaposi sarcoma,
the use of prednisolone in HIV-associated tuberculous pleurisy is not recommended.”
Discussion
The
results of this study show that the use of prednisolone
as an adjunct to treatment for tuberculosis
did not improve survival in the HIV-1 infected adults
with pleural tuberculosis who participated in the study.
There
was a marked and sustained increase in CD4+
T cell counts following treatment for tuberculosis, but, paradoxically, this was accompanied
by an increase in
viral load, suggesting
that the increase in CD4+ T cell counts
may not have been associated with an improvement
in the immune response against HIV infection. A
similar increase in CD4+ T cell counts occurred in both the prednisolone and placebo groups, which is in keeping with prednisolone's lack of effect on mortality.
“Of
great concern,” write the authors, “was the striking
association observed between the use of prednisolone and the incidence of Kaposi sarcoma.” There have been a considerable number of reports of Kaposi sarcoma occurring in patients not infected with
HIV following the use of immunosuppressive
drugs -particularly of glucocorticoids
- after transplantation, in treatment of autoimmune
diseases, and in tuberculous pericarditis.
The
mechanisms by which prednisolone promotes the
development of Kaposi sarcoma remain to be determined.
Kaposi sarcoma
is a disease that causes prolonged suffering,
is difficult and expensive to treat, and is
usually fatal. The investigators therefore conclude,
“In view of the lack of survival benefit and the
significant increase in incidence of Kaposi sarcoma, prednisolone should not be used in the treatment of pleural tuberculosis. This recommendation can
be applied regardless of HIV status, because
no long-term benefits have been reported with regards
to the use of glucocorticoids in patients with
pleural tuberculosis who are not infected with HIV.”
They
continue, “This recommendation cannot necessarily be
generalized to apply to the use of prednisolone
for other forms of tuberculosis or for other
indications in HIV-infected patients. In the
case of pericardial tuberculosis, the use of prednisolone
has been shown to improve survival regardless of
HIV status, and its use should be continued.”
“Similarly, the use of prednisolone is lifesaving, and should continue, in severe Pneumocystis
carinii pneumonia (PCP); however, in HIV-infected patients,
prednisolone should be used with caution for indications
where no beneficial effect on immediate survival can be expected.”
*Pleurisy:
inflammation of the pleura, the membrane that envelops the lungs
and the lining the walls of the pleural cavity.
Uganda Virus Research Institute, Entebbe, and Uganda Case Western Reserve University Research Collaboration, Mulago Hospital, Kampala, Uganda; London School of Hygiene and Tropical Medicine, London, United Kingdom; University of Medicine and Dentistry of New Jersey, Newark.
08/16/04
Reference
A M Elliott
and others. A Randomized, Double-Blind, Placebo-Controlled Trial of the Use of Prednisolone as an Adjunct to Treatment in HIV-1-Associated Pleural Tuberculosis.
The Journal of Infectious Diseases 190(5): 869-878. September 1,
2004.
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