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Therapeutic
Response of HIV-1 Subtype C in African Patients Coinfected with
either Mycobacterium tuberculosis or Human Herpesvirus 8
A
potential confounding factor in the treatment of
HIV infection in Africa is the frequent occurrence
of opportunistic
infections (OIs). OI-induced immune activation
can interfere with HIV-1 clearance by increasing
viral replication and target cell availability.
Treatment
outcomes for patients dually infected with HIV-1
and Mycobacterium tuberculosis
or HIV-1 and human herpesvirus
(HHV)-8 were assessed by measuring changes
in viral load and CD4+
cell counts and by determining the time
taken to reach undetectable HIV-1
RNA levels, assessed by means of Kaplan-Meier
survival analysis.
Patients
with HIV-1 and Kaposi
sarcoma (KS) received generic
nevirapine (Viramune), stavudine
(Zerit), and lamivudine/3TC
(Epivir); patients with HIV-1 and tuberculosis
(TB) received standard commercial didanosine
(Videx), 3TC, and efavirenz
(Sustiva).
Both
cohorts exhibited a rapid, near-exponential phase
I decline in viral load. Patients with TB
and late-stage KS had the steepest decay kinetics.
These same patients had the greatest initial increase
in CD4+ cell counts. Phase II clearance
was slower and more variable.
The
proportions of patients reaching undetectable plasma
HIV-1 levels at days 7, 14, 28, 60, and
90 were, respectively, 15.8%, 30.0%, 52.6%, 78.9%,
and 93.8% (Pearson's 2 =
50.5; P < .001) for patients with TB
and 0.0%, 5.0%, 22.2%, 64.7%, and 80.0% (Pearson's
2
= 63.6; P < .001) for patients with
KS.
Conclusions
The
authors conclude, “Nucleoside reverse-transcriptase inhibitor/non
nucleoside reverse-transcriptase inhibitor based
treatment regimens are highly effective in
clearing rapidly replicating (phase I) virus in
African patients
dually infected with HIV-1 and either TB or
KS.
(See
the editorial commentary by T P Flanigan and
others.).
01/03/04
Reference
E Cassol and others. Therapeutic Response of
HIV-1 Subtype C in African
Patients Coinfected with either
Mycobacterium tuberculosis or
Human Herpesvirus-8. Journal of Infectious Diseases.
December 22, 2004 (online Edition). Scheduled for publication in
JID February 1, 2005.
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